The end of the cellular chromosome is cased by structures called telomeres. Telomere structures stall the disassembling of genes near the ends of the chromosomes by helping chromosome ends to shorten , which is a normal part of the chromosome replication process. With each cell division, telomeres reduce in length, losing the most distal end of the lagging strand.
Dr. Leonard Hayflick identified the way shortening telomeres , will limit the number of times a normal cell population can devide. The reduction in the length of the telomeres limits the cells to a fixed number of divisions. Consumed telomeres indicates a halt to cell division and replication. Further research suggests that this is responsible for aging on the cellular level which translates to a limit on human life span.
Researchers are working to develop a medication or gene therapy that would lengthen the telomeres in selected cells. This would support continued cell division and replication, promoting a longer life for the body. Valuing the connection between health, aging and longevity.
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